下面的Pubmed聯結是我在國防醫學院擔任助教六年的研究足跡 (1989-1995),點進之後最讓我感動的是那一長串的英文: Department of Biochemistry, National Defense Medical Center。第一篇發表於1992年那時我已留校三年重新讀過每篇文章的摘要 (Abstract) 每每讓我想起隱藏在文章背後點點滴滴的故事

對我而言,那是一段非常的歲月,我一輩子都引以為傲能以「National Defense Medical Center」之名發表下面的文章----與學弟妹們共勉。

Purification, cDNA cloning, and expression of human sorcin in vincristine-resistant HOB1 lymphoma cell lines. Lee WP. Arch Biochem Biophys. 1996 Jan 15;325(2):217-26.
http://www.ncbi.nlm.nih.gov/pubmed/8561500

Glutathione S-transferase and glutathione peroxidase are essential in the early stage of adriamycin resistance before P-glycoprotein overexpression in HOB1 lymphoma cells. Lee WP, Lee CL, Lin HC. Cancer Chemother Pharmacol. 1996 Jan;38(1):45-51.
http://www.ncbi.nlm.nih.gov/pubmed/8603451

P-glycoprotein is hyperphosphorylated in multidrug resistant HOB1 lymphoma cells treated with overdose of vincristine. Lee WP. Biochim Biophys Acta. 1995 Aug 17;1245(1):57-61.
http://www.ncbi.nlm.nih.gov/pubmed/7654766

Purification and characterization of tubulin from parental and vincristine-resistant HOB1 lymphoma cells. Lee WP. Arch Biochem Biophys. 1995 Jun 1;319(2):498-503.
http://www.ncbi.nlm.nih.gov/pubmed/7786033

Characterization of vincristine-resistant HOB1 lymphoma cell line showing the classical MDR phenotype and altered expression of membrane glycoproteins. Lee WP, Lee CL. Oncology. 1994 Jan-Feb;51(1):2-8.
http://www.ncbi.nlm.nih.gov/pubmed/7903440

The role of reduced growth rate in the development of drug resistance of HOB1 lymphoma cells to vincristine. Lee WP. Cancer Lett. 1993 Sep 30;73(2-3):105-11.
http://www.ncbi.nlm.nih.gov/pubmed/8221621

Multidrug resistant HOB1 lymphoma cells express P-glycoprotein that does not play the major role in the development of drug resistance to adriamycin. Lee WP, Lee CL, Kao MC. Biochem Biophys Res Commun. 1992 Nov 30;189(1):92-100.
http://www.ncbi.nlm.nih.gov/pubmed/1360209

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